By Philip M. Parker
In March 2001, the nationwide Institutes of well-being issued the subsequent caution: ''The variety of websites providing health-related assets grows on a daily basis. Many websites supply invaluable details, whereas others could have details that's unreliable or misleading.'' moreover, a result of speedy raise in Internet-based info, many hours might be wasted looking out, choosing, and printing. due to the fact that simply the smallest fraction of knowledge facing Amyotrophic lateral sclerosis is listed in se's, corresponding to www.google.com or others, a non-systematic method of web study might be not just time eating, but in addition incomplete. This booklet used to be created for doctors, scholars, and individuals of most of the people who are looking to behavior clinical learn utilizing the main complex instruments to be had and spending the smallest amount of time doing so.
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Extra resources for Amyotrophic Lateral Sclerosis - A Bibliography and Dictionary for Physicians, Patients, and Genome Researchers
The proposed work uses x-ray microbeam (articulatory kinematic), speech acoustic, and speech intelligibility measures to address a major descriptive need, as well as a clinically- and theoretically-relevant hypothesis. Very little is known about lingual behavior in dysarthrias, and what is known about labial and mandibular behavior in dysarthria is confined to a limited type of speech material and measurement strategy. Studies 27 The present proposal includes a variety of speech materials to allow a broad characterization of articulatory kinematics (and the speech acoustic and intelligibility results of those motions) for speech production behavior.
We have proposed a novel mechanism to explain how Zndeficient SOD may cause motor neuron disease and have together coauthored 12 papers on these subjects over the past 7 years. Most recently, we have found that astroglia can play a pathogenic role in motor neuron degeneration. It is well known that reactive astrocytes surround degenerating motor neurons in ALS patients as well as in transgenic mice and rats over-expressing ALS mutant SOD-1. We found that peroxynitrite triggers a long-lasting phenotypic transformation in astrocytes, which promotes apoptosis of motor neurons cultured on the monolayers by mechanisms dependent on nitric oxide (NO) and peroxynitrite formation.
The proteins move in the form of filamentous structures that may represent single neurofilament polymers. Contrary to the widely held view that slow axonal transport is a slow, synchronous and exclusively anterograde movement, we found that the filaments actually move at very fast rates, approaching the rate of fast axonal transport, and that the movements are also infrequent, bidirectional and highly asynchronous. Based on these observations, we have proposed a new model for slow axonal transport in which the actual rate of movement is fast, but the overall rate is slow because the rapid movements are interrupted by prolonged pauses.