By Shanda H. Blackmon, Min P. Kim, Karen J. Dickinson
This atlas presents a finished, cutting-edge evaluation of all interventions that pertain to the esophagus. It contains a evaluate of the present staging modalities, ablation applied sciences, resection and reconstruction options, and ailment category. Evidence-based directions relating to how each one intervention is selected also are integrated. With colour illustrations and images for every surgical procedure, the atlas information particular anatomic themes comparable to micro-anatomy of Barrett’s and Dysplasia, EMR pathology, endoscopic ultrasound, and traditional surgical anatomy. each one intervention is gifted in activity layout as a job checklist to be checked-off as every one step is completed.
Written through specialists within the box, Atlas of Esophageal sickness and Intervention: A Multidisciplinary Approach serves as a helpful source for any practitioner who plays esophageal intervention and may consultant new surgeons and gastroenterologists into the hybrid multidisciplinary method of this disease.
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Extra info for Atlas of Esophageal Disease and Intervention: A Multidisciplinary Approach
Compare the normal histology in Fig. ) Fig. 19 Stricture of the esophagus, fresh specimen 44 Fig. 20 Histology of stricture (H & E, ×20) B. Gorman 2 Histologic Anatomy 45 Inlet Patch The inlet patch is characterized by ectopic gastric mucosa that is surrounded by normal squamous mucosa (Fig. 21). Inlet patches are most frequently seen in biopsy specimens taken from the upper third of the esophagus. a b Fig. 21 (a) Inlet patch (H & E, ×40). The mucosa on the left side of this photomicrograph shows essentially normal gastric mucosa with foveolar and glandular epithelium.
A) Barrett’s esophagus (H & E, ×100). (b) Barrett’s esophagus (H & E, ×40) 27 2 Histologic Anatomy New, more superficial muscularis mucosa Original, deeper muscularis mucosa b Fig. 6 (continued) 28 B. Gorman Fig. 7 (a) Barrett’s esophagus, indefinite for dysplasia (H & E, ×100). (b) Barrett’s esophagus, indefinite for dysplasia (H & E, ×400). The cells show cytologic features of dysplasia that include nuclear enlargement, hyperchromasia, and some nuclear stratification. Diagnostic features of high-grade dysplasia are not seen a b Nuclear enlargement, hyperchromasia and focal stratification Goblet cell Mitotic figure approaching gland lumen 2 Histologic Anatomy 29 Barrett’s Esophagus, Low-Grade Dysplasia Low-grade dysplasia is characterized by nuclear crowding, stratification, atypia, and increased mitotic figures (Fig.
Gorman Glycogen Accumulation Glycogen accumulation creates cytoplasmic clearing in squamous cells (Fig. 24). This finding may mimic the perinuclear halo seen with the human papillomavirus cytopathic effect. The absence of characteristic nuclear changes supports a diagnosis of glycogen accumulation. a b Fig. 24 (a) Glycogen accumulation (H & E, ×100). (b) Glycogen accumulation (PAS without diastase, ×100). The PAS stain highlights the cytoplasmic glycogen 2 Histologic Anatomy 49 Herpes Esophagitis Herpesvirus infects the squamous epithelium of the esophagus, leading to ulceration (Fig.