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By M. Korbonits

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Extra info for Obesity and Metabolism (Frontiers of Hormone Research)

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Certainly, replication in more cohorts and the elucidation of the underlying mechanism remain mandatory. Nevertheless, the discovery of previously unsuspected genes opens up new directions and potentially important insights into the pathogenesis of common diseases. Perspectives: Interactions Even though some recent approaches have been successful in identifying important genetic contributors to obesity, these new genes are obviously not much more than just a few pieces of the puzzle in the complex framework of pathogenic factors contributing to obesity and it is obvious that many more genes await discovery.

N Engl J Med 1988;318:1217–1225. 32 Hanson RL, Ehm MG, Pettitt DJ, Prochazka M, Thompson DB, Timberlake D, Foroud T, Kobes S, Baier L, Burns DK, Almasy L, Blangero J, Garvey WT, Bennett PH, Knowler WC: An autosomal genomic scan for loci linked to type II diabetes mellitus and body-mass index in Pima Indians. Am J Hum Genet 1998;63:1130–1138. 33 Norman RA, Tataranni PA, Pratley R, Thompson DB, Hanson RL, Prochazka M, Baier L, Ehm MG, Sakul H, Foroud T, Garvey WT, Burns D, Knowler WC, Bennett PH, Bogardus C, Ravussin E: Autosomal genomic scan for loci linked to obesity and energy metabolism in Pima Indians.

Diabetes Care 2006;29: 2489–2497. 45 Masud S, Ye S: Effect of the peroxisome proliferator activated receptor-gamma gene Pro12Ala variant on body mass index: a meta-analysis. J Med Genet 2003;40:773–780. 46 Andrulionyte L, Peltola P, Chiasson JL, Laakso M: Single nucleotide polymorphisms of PPARD in combination with the Gly482Ser substitution of PGC-1A and the Pro12Ala substitution of PPARG2 predict the conversion from impaired glucose tolerance to type 2 diabetes: the STOP-NIDDM trial. Diabetes 2006;55:2148–2152.

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